Environmental Secondhand Smoke Exposure Triggered the Old Man Heart Adaptive Responses

Jia-Ping Wu

 

Affiliations: Research Center for Healthcare Industry Innovation, National Taipei University of Nursing and Health Sciences, Taipei City 11219, Taiwan, R.O.C.

 

*Correspondence author: Jia-Ping Wu, Research Center for Healthcare Industry Innovation, National Taipei University of Nursing and Health Sciences. No. 365, Mingde Rd., Beitou Dist., Taipei City 11219, Taiwan, R.O.C.^ Email: affymax0823@yahoo[.]com[.]tw

 

Citation: Jia-Ping Wu (2019) Environmental Secondhand Smoke Exposure Triggered the Old Man Heart Adaptive Responses. Med Healthcare Rep, 1(1);1-9.

 

Copyright: © 2019, Jia-Ping Wu, This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International License, which permits unrestricted use, distribution and reproduction in any medium, provided the original author and source are credited.

 

ABSTRACT:

 

Secondhand Smoke (SHS) exposure has been linked harmful health outcome which is an important cause of short-lifespan morbidity and mortality. However, it is not clear pathological condition in old man exposure to SHS. This report reviews SHS exposure to old age to determine health mechanisms of the heart. SHS exposure increases the primary progressive atherosclerosis and arterial stiffness and increase risk of coronary disease events causes human diseases, especially in elderly. Aging is a physiology process involving progressive impairment of normal heart functions, due to an increasing vulnerability which reduces the ability of survive. Aging of the very elderly heart is associated with heart failure, expected or normal aging change. SHS exposure involves the combination of the smoke emitted by the burning end of a tobacco cigarette and the smoke exhaled by the smoker into the environment. SHS led to cardiac remodeling has been observed in exposed increases cardiovascular diseases mortality. Even for the elderly exposed to SHS at home was higher than outside of the home, or both at home and outside of the home. Experimental evidence in animal models has indicated attenuation in cardioprotective pathways with aging, yet information regarding myocardial dysfunction in elderly age is limited. Therefore, the numerous molecular and biochemical changes also affect the expression levels of human aging cardiac mitochondrial complex phenotype.

 

KEYWORDS: Secondhand smoke, Coronary disease, Cardiovascular diseases, Cardio protective pathways, Myocardial dysfunction, Molecular and Biochemical changes, Myocardial dysfunction

 

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